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Înțelesul "HPV" în dicționarul Engleză HPV - Definiția și sinonimele HPV în dicționarul Engleză Traducerea «HPV» în 25 de limbi human papilloma virus - Traducere în română - exemple în engleză Reverso Context Human papillomavirus hpv high-risk types Human papillomavirus 52 positive squamous cell carcinoma of the conjunctiva The virus infects basal epithelial cells of stratified squamous epithelium.

HPV E6 and E7 oncoproteins are the critical molecules in the process of malignant tumour formation.

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Interacting with various cellular proteins, E6 and E7 influence fundamental cellular functions like cell cycle regulation, telomere maintenance, susceptibility to apoptosis, warts removal treatment adhesion and regulation of immune responses. High-risk E6 and E7 bind to p53 and pRb and inactivate their functions with dysregulation of the cell cycle. Uncontrolled cell proliferation leads to increased risk of genetic instability.

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Lista principalelor căutări efectuate de utilizatori pentru accesarea dicționarului nostru online înEngleză și cele mai întrebuințate expresii cu cuvântul «HPV».

Human papillomavirus results in acestuia se bazează pe analizarea frecvenței de apariție a termenului «HPV» în sursele digitalizate tipărite în Engleză între anul și până în prezent. Intraductal papilloma malignant transformation, it takes decades for cancer to develop.

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This review presents the main mechanisms of HPV genome in the carcinogenesis of the uterine cervix. Virusul infectează epiteliile bazale, celule de epiteliu scuamos stratificat.

Testul HPV va putea fi solicitat într-un interval de 3 săptămâni de la data recoltării probei pentru citologia cervico-vaginală, flaconul Sure Path fiind menţinut în această perioadă în laborator în condiţii de refrigerare. Testul HPV efectuat pe platforma automată Cobas Roche Diagnostics detectează printr-o metodă real-time PCR 14 genotipuri HPV cu risc crescut cu identificarea concomitentă a genotipurilor 16 şi 18 alături de grupul celorlalte 12 tipuri oncogene: 31, 33, 35, 39, 45, 51, 52, 56, 58, 59, 66 şi Pe de altă parte, în comparaţie cu testul HPV standard, noul test prezintă avantajul identificării separate a genotipurilor HPV 16 şi 18 în cadrul aceleiaşi rulări a probei, ceea ce aduce o human papillomavirus results in suplimentară în management-ul pacientelor  cu ASC-US prin detectarea femeilor care prezintă riscul cel mai mare de a dezvolta leziuni premaligne.

Proteinele celulare E6 și E7 influențează fundamental funcțiile celulare, cum ar fi reglarea ciclului celular, întreținerea telomerilor, susceptibilitatea la human papillomavirus hpv high-risk types, adeziunea intercelulară și reglarea răspunsurilor imune. E6 și E7 cu grad ridicat de risc se leagă la p53 și PRB și inactivează funcțiile lor cu dereglarea ciclului celular. Proliferarea necontrolată a celulelor conduce la un risc crescut de instabilitate genetică.

Conținutul Pentru majoritatea oamenilor, virusul papilomavirus uman HPV dispare de la sine, în mod spontan. Dar pentru alții care nu elimină virusul, anumite tipuri de HPV pot provoca cancere cervicale, vaginale și vulvare la femei și cancer anal la bărbați și femei. Alte tipuri pot provoca veruci genitale atât la bărbați, cât și la femei.

De obicei, este nevoie de zeci de ani pentru a dezvolta un cancer. Acest review prezintă principalele mecanisme ale genomului HPV în carcinogeneza colului uterin.

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The most important risk factor in the ethiology of cervical cancer is the persistent infection with a high-risk strain of human papillomavirus. Implicarea genomului papiloma virusului uman hpv în oncogeneza cancerului cervical Materials and methods This general review was conducted based on the AngloSaxone literature from PubMed and Medline to identify the role of HPV genome in the development of cervical cancer.

Discussions Genital human papillomavirus HPV is the most common sexually transmitted infection. Although the majority of infections cause no symptoms and are self-limited, persistent infection with high-risk types of HPV is the most important risk factor for cervical cancer precursors and invasive cervical cancer.

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The presence of HPV in They are human papillomavirus results in responsible for others genital neoplasias like vaginal, vulvar, anal, and penian. HPV is a non-enveloped, double-stranded DNA virus from the family of Papillomaviridae, with an 8 kb circular genome composed of six early ORFs open reading frames with human papillomavirus hpv high-risk types in viral human papillomavirus results in and replication E1, E2, E4, E5, E6, E7two late ORFs L1,2-capsid proteins and a non-coding long controlled region LCR that contains a variety of cis elements, which regulate viral replication and gene expression.

More than HPV types have been identified, and about 40 can infect the genital tract.

Aceste exemple pot conține termeni colocviali. Traducere "HPM - Virusul Papiloma uman" în engleză Alte traduceri Evident, au fost aplicaţii practice incredibile asociate cu lumea aceasta - cum ar fi eradicarea variolei, introducerea vaccinului împotriva cancerului cervical, despre care ştim acum că este produs de HPM - Virusul Papiloma uman. And obviously there's been tremendous practical applications associated with this world - things like the eradication of smallpox, the advent of a vaccine against cervical cancer, which we now know is mostly caused by human papillomavirus.

Based on their association with cervical cancer and precursor lesions, HPVs are grouped to high-risk 16, 18, 31, 33, 34, 35, 39, 45, 51, 52, 56, 58, 59, 66, 68, 73, 82 and low-risk HPV types 6, 11, 42, 43, human papillomavirus hpv high-risk types, 54, 61, 70, 72, Natural history Most genital HPV infections are benign, subclinical, and self-limited, and a high proportion of infections associated with low-grade cervical dysplasias also regress spontaneously 1.

Department of Ophthalmology, Grigore Human papillomavirus results in.

Human papillomavirus results in

By contrast, persistent cervical infection infection detected more than once in an malign cancer tumor of 6 months or longer with an oncogenic HPV type, especially HPV 16 and HPV 18, is the most important risk factor for human papillomavirus results in to high-grade dysplasia, a precancerous lesion that should be treated to prevent the development of invasive cancer 2.

HPV is a necessary but not a sufficient condition for the development of cervical cancer.

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Cofactors associated with cervical cancer include: cigarette smoking, increased parity, increased age, other sexually transmitted infections, immune suppression, long-term oral contraceptive use, and other host factors.

Figure 1. Involvement of Human Papillomavirus genome in oncogenesis of cervical cancer Schematic representation of the HPV double-stranded circular DNA genome Journal of Virology Nov HPV integration into the host genome and Papillomavirus life cycle To establish infection, the virus must infect basal epithelial cells of stratified squamous epithelium, that are long lived or have stem cell-like properties.

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  • Human papillomavirus 52 positive squamous cell carcinoma of the conjunctiva

Microtrauma of the suprabasal epidermal cells enables the virus to infect the cell within the basal layer. Once inside the host cell, HPV DNA replicates as the basal cells differentiate and progress to the surface of the epithelium.

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The viral genome maintains itself as an episome in basal cells, where the viral genes are poorly expressed. In the differentiated keratinocytes of the suprabasal layers of the epithelium, the virus switches to a rolling-circle mode of DNA replication, amplifies its DNA to high human papillomavirus hpv high-risk types number, synthesizes capsid proteins, and causes viral assembly to occur 3.

Their function is to subvert the cell growth-regulatory pathways by binding and inactivating tumor suppressor proteins, cell cyclins, and cyclin-dependent kinases and modify the cellular environment in order to facilitate viral replication in a cell that is terminally differentiated and has exited the cell cycle 4.

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Cell growth is regulated by two cellular proteins: the tumor suppressor protein, p53, and the retinoblastoma gene product, pRB. Unlike in many other cancers, the p53 in cervical cancer is usually wild type and is not mutated.

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E6  binds to p53 via a cellular ubiquitin ligase named E6AP, so human papillomavirus hpv high-risk types it becomes ubiquitinated, leading to degradation and down-regulation of pathways involved in cycle human papillomavirus hpv high-risk types  and apoptosis.

This degradation has the same effect as an inactivating mutation.